Whenever potential, specimens for these checks must be obtained earlier than initiating antimicrobial therapy, as a result of earlier therapy with antimicrobial brokers makes isolation by tradition unlikely. In reality, reentry typically just isn't the mechanism of arrhythmia development in these medical instances: the arrhythmias may be attributable to delayed after-depolarizations, and the triggered activity may be mediated via catecholamines (Lerman et al. Brit Med J management of ascites, spontaneous bacterial peritonitis, 1971;4:266-270
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